ccnmd pitt.edu

Conte Center for Translational Mental Health Research David A. Lewis, MD, Director

News, Events and Outreach. To answer this question, complementary studies in the Center will test the following central hypothesis. Cell type-autonomous molecular disturbances in layer 3 pyramidal cells of the neocortex give rise to morphological abnormalities in these neurons, the severity of which is moderated by normal differences across cortical regions. This pathology alters cortical circuitry within and between regions, impairs functional connectivity and neural synchrony across regions, and d.

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Conte Center for Translational Mental Health Research David A. Lewis, MD, Director

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News, Events and Outreach. To answer this question, complementary studies in the Center will test the following central hypothesis. Cell type-autonomous molecular disturbances in layer 3 pyramidal cells of the neocortex give rise to morphological abnormalities in these neurons, the severity of which is moderated by normal differences across cortical regions. This pathology alters cortical circuitry within and between regions, impairs functional connectivity and neural synchrony across regions, and d.

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The web site ccnmd.pitt.edu states the following, "To answer this question, complementary studies in the Center will test the following central hypothesis." We noticed that the website said " Cell type-autonomous molecular disturbances in layer 3 pyramidal cells of the neocortex give rise to morphological abnormalities in these neurons, the severity of which is moderated by normal differences across cortical regions." It also said " This pathology alters cortical circuitry within and between regions, impairs functional connectivity and neural synchrony across regions, and d."

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